Razlika između verzija stranice "Treća moždana komora"

U nekoliko istraživanja pronađeni su dokazi da je proširenje moždanih komora može biti povezano s velikim [[biologija depresije|depresijijama]], a posebno proširenjem treće komore.<ref>{{Cite journal|first1=C.A.|last1=Hendrie|first2=A.R.|last2=Pickles|title=Depression as an evolutionary adaptation: Implications for the development of preclinical models|url=http://www.academia.edu/1145742/Depression_as_an_evolutionary_adaptation_Implications_for_the_development_of_preclinical_models|journal=Medical Hypotheses|year=2009|volume=72|pages=342–347|accessdate=25. 9. 2013|doi=10.1016/j.mehy.2008.09.053|pmid=19153014}}</ref><ref>{{Cite journal|first1=C.A.|last1=Hendrie|first2=A.R.|last2=Pickles|title=Depression as an evolutionary adaptation: Anatomical organisation around the third ventricle|url=http://www.academia.edu/1145735/Depression_An_Evolutionary_Adaptation_Organised_Around_the_Third_Ventricle|journal=Medical Hypotheses|year=2010|volume=74|pages=735–740|accessdate=25. 9. 2013|doi=10.1016/j.mehy.2009.10.026|pmid=19931308}}</ref><ref>{{Cite journal|first=Yvette|last=Sheline|title=Neuroimaging studies of mood disorder effects on the brain|url=http://www.biologicalpsychiatryjournal.com/article/S0006-3223%2803%2900347-0/abstract|journal=Biological Psychiatry|volume=54|pages=338–352|date=Augustaugust 2003|accessdate=25. 9. 2013|doi=10.1016/s0006-3223(03)00347-0|pmid=12893109}}</ref> These observations are interpreted as indicating a loss of neural tissue in brain regions adjacent to the enlarged ventricle, leading to suggestions that [[cytokine]]s and related mediators of [[neurodegeneration]] may play a role in giving rise to the disease.<ref>{{Cite journal|first1=Husseini K.|last1=Manji|first2=Jorge A.|last2=Quiroz|first3=Jonathan|last3=Sporn|first4=Jennifer L.|last4=Payne|first5=Kirk|last5=Denicoff|first6=Neil A.|last6=Gray|first7=Carlos A.|last7=Zarate Jr.|first8=Dennis S.|last8=Charney|title=Enhancing neuronal plasticity and cellular resilience to develop novel, improved therapeutics for difficult-to-treat depression|url=http://www.biologicalpsychiatryjournal.com/article/S0006-3223%2803%2900117-3/abstract|volume=53|pages=707–742|date=Aprilapril 2003|accessdate=25. 9. 2013}}</ref><ref>{{cite journal | last1 = Miller |first1=A. H.|last2= Maletic|first2= V. |last3=Raison |first3=C. L. | year = 2009 | title = Inflammation and its discontents: the role of cytokines in the pathophysiology of major depression | url = | journal = Biological Psychiatry | volume = 65 | issue = 9| pages = 732–741 | doi=10.1016/j.biopsych.2008.11.029 | pmid=19150053}}</ref><ref>{{cite journal | last1 = Raison |first1=C. L.|last2= Capuron |first2=L.|last3= Miller|first3= A. H. | year = 2006 | title = Cytokines sing the blues: inflammation and the pathogenesis of depression | url = | journal = Trends in Immunology | volume = 27 | issue = 1| pages = 24–31 | doi=10.1016/j.it.2005.11.006 | pmid=16316783 | pmc=3392963}}</ref>